Herpes Research Today is a free monthly online journal that collates and summarizes the latest research about Herpes, including details on herpes simplex virus (hsv), genital, oral, symptoms, treatment.

Volume 2 (2005), Issue 9 (September)

1. 4-Oxo-4,7-dihydrothieno[2,3-b]pyridines as non-nucleoside inhibitors of human cytomegalovirus and related herpesvirus polymerases.
   J Med Chem, 48(18): 5794-804. [Abstract] [Full-text]
2. Effects of substitutions of arginine residues on the basic surface of herpes simplex virus UL42 support a role for DNA binding in processive DNA synthesis.
   J Virol, 79(18): 12025-34. [Abstract] [Full-text]
3. The extracellular domain of herpes simplex virus gE is indispensable for efficient cell-to-cell spread: evidence for gE/gI receptors.
   J Virol, 79(18): 11990-2001. [Abstract] [Full-text]
4. In vivo kinetics of GITR and GITR ligand expression and their functional significance in regulating viral immunopathology.
   J Virol, 79(18): 11935-42. [Abstract] [Full-text]
5. Herpes simplex virus glycoprotein B binds to cell surfaces independently of heparan sulfate and blocks virus entry.
   J Virol, 79(18): 11588-97. [Abstract] [Full-text]
6. Chondroitin sulfate characterized by the E-disaccharide unit is a potent inhibitor of herpes simplex virus infectivity and provides the virus binding sites on gro2C cells.
   J Biol Chem, 280(37): 32193-9. [Abstract] [Full-text]
7. Circularization of the herpes simplex virus type 1 genome upon lytic infection.
   J Virol, 79(19): 12487-94. [Abstract] [Full-text]
8. Reciprocal activities between herpes simplex virus type 1 regulatory protein ICP0, a ubiquitin E3 ligase, and ubiquitin-specific protease USP7.
   J Virol, 79(19): 12342-54. [Abstract] [Full-text]
9. A herpes simplex virus type 1 mutant expressing a baculovirus inhibitor of apoptosis gene in place of latency-associated transcript has a wild-type reactivation phenotype in the mouse.
   J Virol, 79(19): 12286-95. [Abstract] [Full-text]
10. Characterization of VP22 in herpes simplex virus-infected cells.
    J Virol, 79(19): 12185-98. [Abstract] [Full-text]
11. The cellular response to herpes simplex virus type 1 (HSV-1) during latency and reactivation.
    J Neurovirol, 11(4): 376-83. [Abstract] [Full-text]
12. Stress up-regulates neuronal expression of the herpes simplex virus type 2 large subunit of ribonucleotide reductase (R1; ICP10) by activating activator protein 1.
    J Neurovirol, 11(4): 329-36. [Abstract] [Full-text]
13. Amplification of Herpes simplex type 1 and Human Herpes type 5 viral DNA from formalin-fixed Alzheimer brain tissue.
    Neurosci Lett, 390(1): 37-41. [Abstract] [Full-text]
14. A monoclonal antibody-based ELISA allows discrimination between responses induced by bovine herpesvirus subtypes 1 (BoHV-1.1) and 2 (BoHV-1.2).
    J Virol Methods, 129(2): 191-3. [Abstract] [Full-text]
15. A role for herpesvirus entry mediator as the receptor for herpes simplex virus 1 entry into primary human trabecular meshwork cells.
    J Virol, 79(20): 13173-9. [Abstract] [Full-text]
16. Herpes simplex virus tegument protein VP22 contains an internal VP16 interaction domain and a C-terminal domain that are both required for VP22 assembly into the virus particle.
    J Virol, 79(20): 13082-93. [Abstract] [Full-text]
17. Herpes simplex virus 1 envelopment follows two diverse pathways.
    J Virol, 79(20): 13047-59. [Abstract] [Full-text]
18. Virus infection switches TLR-3-positive human neurons to become strong producers of beta interferon.
    J Virol, 79(20): 12893-904. [Abstract] [Full-text]
19. Identification and functional evaluation of cellular and viral factors involved in the alteration of nuclear architecture during herpes simplex virus 1 infection.
    J Virol, 79(20): 12840-51. [Abstract] [Full-text]
20. Site-directed mutagenesis of large DNA palindromes: construction and in vitro characterization of herpes simplex virus type 1 mutants containing point mutations that eliminate the oriL or oriS initiation function.
    J Virol, 79(20): 12783-97. [Abstract] [Full-text]
21. Role of the central amygdala in modulating the pituitary-adrenocortical and clinical responses in experimental herpes simplex virus-1 encephalitis.
    Neuroendocrinology, 81(4): 267-72. [Abstract] [Full-text]

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